Winstrol Doses

When it comes to anabolic steroids, total dosing of a particular steroid can vary dramatically. Take for example, testosterone; the high end dose of Testosterone-Cypionate among HRT patients is generally 200mg per week. For the performance enhancer, 400mg per week is considered the bottom dose of Testosterone-Cypionate, with many going as high as 1,000mg per week, and more than you'd think even higher than that. Then we have steroids like Stanozolol, or as you know it by its most common trade name Winstrol. Winstrol doses are generally not so dramatically spread out. We can tell you right from the get go the average male Winstrol doses range from 50mg-100mg per day for physique minded individuals, and 10mg every other day to every day for females. For those who are supplementing for athletic performance the doses are often even lower. For male athletes, Winstrol doses can be as low as 20-25mg every other day, but for females, the average Winstrol doses will still fall in the 10mg every other day range. It's important to note, especially regarding male athletes, the total doses can be more in-line with physique doses, but these lower doses are not uncommon.

With all this in mind, the most obvious question is how to maximize our Winstrol doses so that we get the most bang for our buck. After all, the Stanozolol hormone, while not the most expensive is definitely not the cheapest. At any rate, that's what we want to discuss today. We'll show you how to plan out your total dosing and use for every type of cycle, both men and women, and then you can decide in Winstrol is for you.

Male Off-Season Winstrol Doses

The Stanozolol hormone is not one of the better off-season steroids; it's simply not a mass builder. However, some will use it for this purpose as it will dramatically lower SHBG and create a nice synergetic effect with other steroids being used. Further, although there is no hard proof, many performance enhancers have reported use during an off-season cycle will help solidify gains, and if this is true it can be a welcomed effect. Even so, we must remember this is a fairly liver toxic steroid, and with that in mind it is best served during other cycles so as not to cause too much undue stress to the liver. In any case, if you decide to use Stanozolol for your off-season cycle you will find it best served at the end. Winstrol doses in the 50mg every other day range for approximately 4-6 weeks will be more than enough to create the effect you're looking for in any man.

Female Off-Season Winstrol Doses
While Stanozolol is not the greatest mass builder for the male performance enhancer, we can't say the same for females. Females are far more sensitive to the steroid, and while it will primarily benefit them in other cycles it will be far more beneficial to females when bulking than compared to men. If the female can tolerate 10mg per day Winstrol doses this will undoubtedly pack on some nice lean tissue, but some may be too sensitive and be forced to keep it at 10mg every other day. At any rate, even at this lower dose, if virilization symptoms begin to show you are encouraged to discontinue use. If you ignore the symptoms, you may find they become a permanent problem.

Male Cutting Winstrol Doses
The cutting period is the best time period for a man to supplement with Winstrol; in-fact, it can be one of the best cutting steroids of all. To obtain a truly lean and granite like physique, Stanozolol can definitely be part of the total answer. Most men will find Winstrol doses of 50mg per day for the last 6-8 weeks of the cycle to be perfect. For those who are competing in a bodybuilding contest, it's not a bad idea to increase the dose to 100mg per day the last 10-14 days before the show. This is not a dose we want to run for a long period of time, for such a dose for weeks on end will cause too much stress to the liver, and if your liver is not healthy you're not healthy.

Female Cutting Winstrol Doses
Without question, Anavar is the ultimate anabolic steroid for women, but Winstrol can be a reasonable choice too. The odds of adverse side-effects are higher with Winstrol than compared to Anavar, but the majority of women who supplement responsibly will be just fine. Women who supplement with this steroid for cutting purposes will receive the same benefits as men. Their physique will be leaner and harder, and they will preserve a lot of tissue and strength that is often lost with hard dieting. To achieve this end, the vast majority of female users will only need Winstrol doses of a 10mg every other day range, with 5-6 weeks of use being more than sufficient.

Athletic Performance Winstrol Doses
By its nature, the Stanozolol hormone can significantly increase strength. For this reason, Winstrol has for decades been a favorite of many athletes; after all, when it comes to sports, strength and speed is the name of the game. Most men who supplement for this purpose will find Winstrol doses of 25mg-50mg every other day to be just about perfect. For the female athlete, 10mg every other day will suffice. For the athlete looking to supplement, such Winstrol doses will provide the boost in strength and speed they're looking for without adding a lot of lean tissue that might be a hindrance, especially as it pertains to prying eyes.

Properties of Winstrol

Winstrol is the trade name for the anabolic steroid Stanozolol. This is the third most popular and widely used anabolic steroid in all history and in the whole world. The first most popular anabolic steroid is Dianabol (Methandrostenolone), second most popular is Nandrolone Decanoate (Deca Durabolin), and the third most popular is Winstrol (Stanozolol).

Half Life: 9 hours (oral), 24 hours (injectable)
Detection Time: 2 months
Anabolic Rating: 320
Androgenic Rating: 30


Studies have demonstrated that Winstrol’s main mechanism of action is that of binding with cellular androgen receptors as opposed to non-receptor mediated activity (such as those possessed by Dianabol or Anadrol). It is also believed that Winstrol also possesses some very small measurable form of anti-Progestogenic properties in regards to the Progesterone receptor, although this is not fully understood. In addition to some small antagonistic effects on the Progesterone receptor, it has been found that Winstrol also possesses low affinity for Glucocorticoid-binding site interactions, as well as activity that is independent of Androgen receptors, Progesterone receptors, and Glucocorticoid receptors. Winstrol has not been found to have any notable Progestogenic activity in the body as well.

Winstrol possesses a very high binding affinity for SHBG (Sex Hormone Binding Globulin), therefore granting far more of Winstrol (as well as other anabolic steroids that may be stacked alongside it, such as Testosterone) to freedom in the bloodstream in doing its job of signaling muscle growth. SHBG is a protein that attaches and binds to other sex hormones (Testosterone, Estrogen, or any synthetic anabolic steroid) and renders them useless as long as SHBG is bound to that hormone. Effectively, SHBG places ‘handcuffs’ on any hormone it binds to and prevents it from doing its job. Winstrol has also demonstrated to not only prevent SHBG from binding with other anabolic steroids, but it has also demonstrated strong suppression of SHBG production in the body. For example, one particular study conducted on 25 male test subjects where Winstrol was administered orally resulted in a 48.4% drop in SHBG levels following just 3 days of Winstrol administration.

With Winstrol being a DHT-derivative, it holds the advantage that is generally associated with DHT and all other DHT-derivatives: it is unable to bind with the aromatase enzyme, which results in no possible Estrogen conversion. Resulting from this is an avoidance of the Estrogen-related side effects of water retention (and the associated risks of elevated blood pressure), as well as other Estrogen-related side effects. Being a DHT-derivative, it is also unable to interact with the 5-alpha reductase enzyme, which is the enzyme responsible for the conversion of Testosterone into Dihydrotestosterone. As Winstrol is already a modified form of DHT, this cannot possibly occur.

Winstrol exhibits a longer half-life as a result of its structural modifications, enabling the injectable format of Winstrol to possess a half-life of approximately 24 hours, and 9 hours for the oral preparation of Winstrol. In relation to Testosterone, Winstrol holds an androgenic strength rating of 30 with an anabolic strength rating of 320, which is quite significant considering this means Winstrol is slightly over three times the anabolic strength of Testosterone. In order for any individual to understand the meaning of these numbers and ratings, it must be understood that the base reference measurement for these strength ratings is the number one anabolic steroid Testosterone. Testosterone is utilized as the measuring stick or the measuring bar whereby all other anabolic steroids are referenced with and compared to (much like the celcius scale of temperature measurement where the freezing point and boiling points of water is used as the baseline measurement for temperature). Upon understanding this, any individual can easily observe how Winstrol possesses an anabolic strength of three times Testosterone (Testosterone’s anabolic and androgenic ratings are both respectively 100). Percentage-wise, it could be described that Winstrol is 320% more anabolic than Testosterone, and it is 30% less androgenic than Testosterone.

An important fact that must be reminded to the reader is the fact that both the injectable and oral preparations of Winstrol possess the exact same chemical structure. This is unlike nearly all other anabolic steroids, where oral preparations are always C17-alpha alkylated, and injectable preparations are absent of this methylation (and often injectable compounds are also esterified to modulate the release rate and half-life). This is not so with Winstrol, where the oral and injectable preparations are exactly 100% identical to each other. This presents some concerns that the reader must be aware of: The result is a greater amount of hepatotoxicity (liver toxicity), and because both the injectable and oral preparations both possess the hepatotoxic modification of C17-alpha alkylation, they both will place an almost equal level of hepatotoxic strain on the liver. However, the injectable preparation avoids the first-pass through the liver, which allows it to be slightly less hepatotoxic than the oral Winstrol preparation – but hepatotoxic nevertheless, and its duration of use must also have limitations placed on it.

The best estrogen maintenance drug

Formestane is the first in a new line of selective, steroidal-based aromatase inhibitors. It is currently available in a few European countries, and is expected to reach U.S. shelves before long. Formestane is structurally a derivative of androstenedione, most specifically 4-OH androstenedione. Androstenedione is a readily aromatized steroid, so clearly this similarity welcomes interaction with the aromatase enzyme. Its activity in the body is in fact that of a suicide inhibitor, meaning that the compound will become inextricably bound to the aromatase enzyme upon contacting it. Its effect is therefore comparatively much more lasting than that seen with reversible, competitive inhibitors. This is no doubt the reason formestane was shown in studies to be as much as 60 times more potent than aminoglutethimide. As with Arimidex, this compound can help achieve near total suppression of aromatase.
Due to poor oral bioavailability, formestane is commercially prepared as an injectable product. The typical recommendation is an injection of 250mg (typically 1 ampule or injection) every two weeks. Though estrogen suppression can be marked with this dosing pattern, some studies do suggest that by the second week (near the time the dosage is to be repeated) estrogen levels may begin to recover.
The BEST estrogen maintenance drug?
Clearly there is more to consider in choosing an estrogen maintenance drug than just which is the most effective. The discussed differences in cholesterol alterations between anti-estrogens and aromatase inhibitors for example are worth taking into account. In this regard an antiestrogen such as tamoxifen or clomiphene would be most preferred. This is in great contrast to aminoglutethimide, Nolvadex or Clomid, which can cost $100 or less monthly. By measure of which is the most efficient remedy for estrogenic side effects, it would seem that Arimidex and formestane would technically be the most effective. Though more expensive, formestane was actually shown to be slightly less reliable than Arimidex in head to head studies, so its higher price should not automatically lead us into thinking it is the superior of the two. Ultimately however, it is doubtful that either of the new selective aromatase inhibitors (or other related agents slated to be released) will prove to be leagues ahead of the already tried and accepted estrogen maintenance drugs Nolvadex, Clomid and aminoglutethimide in the eyes of athletes. This is simply because the mentioned agents all deal with the action/buildup of estrogen quite effectively (in terms of clinical effectiveness of three agents compare closely to the new selective inhibitors), and outside of a medical setting the high cost of these newer agents will likely prohibit wide spread use.

Bodybuilding And Creatine

Creatine (methylguanidine-acetic acid) was discovered in 1832 by Michel Eugene Cheverul. Later on, in 1834 Justus von Lieburg “confirmed” that creatine was a normal part of meat. It was also found that there was more creatine in wild animals which underwent more exercise than animals that were living in captivity which exercised less.
During the early part of the 1900s by using creatine as a supplement allowed for a boost in creatine in animals. Later on, phosphocreatine (creatine phosphate or phosphorylated creatine) was discovered in the year of 1927. Then in 1934, the creatine kinase (the enzyme that “catalyzes” phosphocreatine was found). Finally, in 1968, phosphocreatine was found in the process of recuperating from exercise.
In foods, creatine is found primarily in red meat and fish. Eaten creatine is then eventually sent to the bloodstream. Creatine is also synthesized within the body by the liver, kidney and pancreas, although this primarily takes place in the liver. This is done in two steps: the first step is when an amidine group from arginine goes to glycine to make guanidinoacetic acid. Then in step two, a methyl group goes to a guanidinoacetic acid from S-adenoslymthionine forming creatine. In the synthesis of creatine, there are some controls on it so that when there is less creatine in one’s diet, there will be more synthesis of creatine in the body. In opposition, if there is a lot of creatine present in one’s diet, then there will be less creatine synthesis in the body.
The storage of creatine in the body occurs in two forms; in the form of phosphocreatine or simply creatine. In the average adult male weighing 70kg, there is 120g of creatine of which 95% is found in the skeletal muscle. Some of the creatine goes to other various parts of the body such as the heart and brain. Of all the creatine in the skeletal muscles, 60-70% of that creatine is phosphocreatine. And because it is phosphocreatine, it cannot leave the membranes.